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Info. Detail :
Curriculum Vitae of Professor Jun Wu
english.swhospital.com 2009-09-10

Personal Information:
Name: Jun Wu
Date of Birth: February 27, 1962
Birth Place: Chongqing
Sex: Male
Marital Status: Married
Tel/Fax: 0086-23-65461677
E-mail: junwupro1@yahoo.com.cn

 

Education:
● 1979.07-1984.07 Department of Clinical Medicine, the Third Military Medical University, bachelor’s degree of medicine.
● 1984.09-1987.07 Graduate student majoring in pathology, the Third Military Medical University, master’s degree of medical pathology
 
Work experience:
● Assistant, then instructor of Department of Pathology, the Third Military Medical University
● Visiting Research Fellow engaging in immunology of transplantation, in Academy of Medicine, University of Verona, Italy
● 1995.02-present  Research Fellow engaging in wound healing and transplantation immunology in the Institute of Burn Research, Southwest Hospital, the Third Military Medical University; Supervisor of master and doctor student.
 
Present post and rank:
● Vice president of Southwest Hospital, Third Military Medical University
● Vice chairman of Chinese Doctor Society of Burn Injury
● Vice chairman of the Organ Transplantation Committee of PLA
● Chairman of Chongqing Organ Transplantation Society, China
● Vice chairman of Chongqing Bioengineering Society, China
● Commissioner of Society of Organ Transplantation, Chinese Medical Association
● Executive member of Society of Burn Injury and Plastic Surgery, Chinese Medical Association
● Director of Chongqing Key Lab for Diseases Proteomics 

Research area
1.Transplantation immunology
2.Wound healing and biomaterials
3.Diseases proteomics 
 
Achievements
● Second-class prize winner of the National Science and Technology Advancement, 2006
● Second-class prize winner of the Science and Technology Advancement of PLA, 2003
● Williams Prize winner of Isreal Burn Society, 1992

Fundings
● Graft Sustenance System. 300,000RMB The Scientific and Technological Committee of Chongqing 2008
● Construction of Chongqing Key laboratory of Proteomics of Disease. 1,000,000RMB The Scientific and Technological Committee of Chongqing 2006
● The role of the interaction between P311 and ITGB4BP in the trans-differentiation of myofibroblast. 280,000RMB The National Natural Science Funds 2007
● Study of the key technology of chitin and animal-derived material tissue engineering skin. 8,000,000RMB The National High Technology Research and Development Project of China (863 Project)(No. 2006AA02A121) 2007
● The regulatory roles of ITGB4BP in hypertrophic scar formation. 900,000RMB National Key Lab Fundation 2008
● Gene modified pig skin as the biodressing for burn wounds. 12,000,000RMB National Dev&Ref Dept Industrial Fundation 2005 
 
Publications in recent years
1.Analyses of differential proteome of human synovial fibroblasts obtained from arthritis. Clinical Rheumatology 2009, 28:191-199 (correspondence author)
2.Activated T cells inhibit NK cell-mediated tumor rejection. Scandinavian Journal of Immunology 2009, 69: 337-341 (correspondence author) 
3.Tolerant T cells inhibit NK cells function in antigen-presenting cells independent way. Transplantation Proceedings 2009, 41: 1844-1847 (correspondence author).
4.Detection of Urinary Biomarkers for Early Diagnosis of Acute Renal Allograft Rejection by Proteomic Analysis. Proteomics – Clinical Application 2009, 3: 694-704 (correspondence author)
5.A novel recombinant immunotoxin with the smallest ribosome-inactivating protein Luffin P1: T cell cytotoxicity and prolongation of allograft survival. Journal of Cellular and Molecular Medicine 2009 (Accepted) (correspondence author)
6.Fluid resuscitation for major burn patients with the TMMU protocol. Burns 2009 (on line) (correspondence author)
7.Unique proteomic features induced by a potential antiglioma agent, Nordy(di-nordihydroguaiaretic acid), in glioma cells. Proteomics 2008, 8:484-494
8.Antibacterial effect of dressings containing multivalent silver iron carried by zirconium phosphate on experimental rat burn wounds. Wound Repair and Regeneration 2008, 16: 800-804
9.hCTLA4-gene modified human bone marrow derived mesenchymal stem cells as allogeneic seed cells in bone tissue engineering. Tissue Engineering 2006; 12(9):2583-2590 (correspondence author)
10.Delivering PD-1 Inhibitory Signal Concomitant with Blocking ICOS Co-Stimulation Suppresses Lupus-Like Syndrome in Autoimmune BXSB Mice. Clin. Immunol. 2006;118:258-67.
11.CTLA4Ig introduced by Adenovirus vector locally to prolong the survival of xenoskin graft on rat burn wound. J Trauma 2005; 59:1209-15 (correspondence author)
12.The role of NO in macrophage dysfunction at early stage after burn injury. Burns 2005, 31:138-144 (correspondence author)
13.Gene Expression of Early Hypertrophic Scar Tissue Screened by Means of cDNA Microarrays. J Trauma 2004, 57: 1276-1286 (first and correspondence author)
14.Effect of early escharectomy and skin graft on immune cells of severe burn patients. J Trauma Surg. 2004, 6:91-93 (correspondence author)
15.Prolongation of survival of alloskin grafts with no concurrent general suppression of the burned patient’s immune system: a preliminary clinical investigation. Burns, 1996, 22(5): 353  (first and correspondence author)
16.An investigation into the mechanisms by which the human dermis does not significantly partake to the rejection of allo-skin grafts. Burns 1995, 21(1): 11 (first and correspondence author)
17.Lack of relationship between the immunologic of the lymphocytic clusters formed in microtray wells.J. Exp. Clin. Cancer Res. 1995, 14: 45 (first author)
18.Donor’s and recipient’s antigen presenting cells cooperatively enhance the allo-rreactive proliferation of peripheral blood lymphocytes. Life Sci. 1994, 54(14): 1009 (first and correspondence author)
19.A preferential homing of lymphocytes into the epidermis causes the inertia of dermis in allo-skin immune rejection.FASEB J. 1994, 8: A449 (first author)
20.Beta2-microglobulin-dependent positive cooperation between antigen-presenting cells.In Vitro Cell Developm. Biol.1994, 30A: 100 (first author)
21.A β2m-dependent mechanism enhances the DNA-synthetic response of recipient’s lymphocytes to the donor’s cells.FASEB J.1994, 8: A679 (first author)
22.A novel approach to extend the survial of skin xenografts without entailing general immunosuppression. Burns 1993, 19: 289 (first and correspondence author)
23.The in utero initiation with DMN alters the complement of cytosolic glutathione S-transterases and the Phenobarbital-induced expression of c-jun oncogenes in primary neonatal rat hepatocytes, Cytotechnology 1993, 11: S18 (first author)
24.A β2m-McAb prevents the reactive proliferation of lymphocyte elicited by allo-human epidermal cells. J. Immuological Research 1992, 4: 21 (first and correspondence author)
25.GST-P expression and in vitro promotion of neonatal rat hepatocytes initiated in  utero with DMN.In Vitro Cell Developm. Biol. 1992, 28: 150A (first author)
26.PTH1-34 induces the differentiation program in adult human keratinocytes cultured in MCDB153 synthetic medium.Mol. Biol. Cell 1992, 3: 176A  (first author)
27.Blood monocots partake to the presentation of allo-epidermal cell antigens to peripheral blood lymphocyte in mixed skin cell-lymphocyte reactions. J. Cell Biol. 1991,115:239A (first author) 

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